p. 191−196
2345-3729
Vol.15/No.4
p. 197−204
2345-3729
Vol.15/No.4
0.05). There was no statistically significant “time trend” for C-reactive protein and TNF-α (P>0.05). Considering both group effect and Time effect, the changes were not significantly different for CRP (P= 0.86) and TNF-α (P=0.69). In contrast, the clinical score and the clinical pulmonary infection score (CPIS) 6, had 100% specificity for diagnosing VAP. With respect to prognosis, only an unchanged or decreasing TNF-α from day 1 to day 3 was marginally associated with 28- day survival. However, day 1 and day 3 acute physiology and chronic health evaluation II (APACHE II) scores were highly associated with 28-day survival. Conclusion: Unlike clinical scoring system including CPIS and APACHE II, TNF-α and CRP levels were not useful as diagnostic or prognostic biomarkers for differentiating between SIRS with VAP etiology and SIRS without infectious etiology.]]>
p. 205−212
2345-3729
Vol.15/No.4
p. 213−217
2345-3729
Vol.15/No.4
p. 218−224
2345-3729
Vol.15/No.4
p. 225−235
2345-3729
Vol.15/No.4
p. 236−242
2345-3729
Vol.15/No.4
p. 243−245
2345-3729
Vol.15/No.4
p. 246−248
2345-3729
Vol.15/No.4
p. 249−250
2345-3729
Vol.15/No.4